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Repurposing of semaglutide by targeting SIRT1 and TGF‐β/Smad signaling in hepatic fibrosis | Naunyn-Schmiedeberg's Archives of Pharmacology
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The weight-loss-independent hepatoprotective benefits of semaglutide are orchestrated by intrahepatic sinusoidal endothelial GLP-1 receptors: Cell Metabolism
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The weight-loss-independent hepatoprotective benefits of semaglutide are orchestrated by intrahepatic sinusoidal endothelial GLP-1 receptors: Cell Metabolism
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Cholestasis: An impairment in bile secretion or flow caused by intrahepatic or extrahepatic disease. Cholestasis is biochemically characterized by increased serum alkaline phosphatase and bilirubin. Clinical symptoms include jaundice (serum total
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Repurposing of semaglutide by targeting SIRT1 and TGF‐β/Smad signaling in hepatic fibrosis | Naunyn-Schmiedeberg's Archives of Pharmacology
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Modulation of metabolic, inflammatory and fibrotic pathways by semaglutide in metabolic dysfunction-associated steatohepatitis
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